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Iodine and Gastric Cancer, Stomach Cancer, 30TH ANNUAL MEETING OF EUROPEAN THYROID ASSOCIATION

17th September 2007 by Arrow Durfee Posted in Uncategorized

30TH ANNUAL MEETING OF EUROPEAN THYROID ASSOCIATION
Istanbul, Turkey, 18-22 September 2004
P.111 (Abstract No: 257)
IS IODINE PROPHYLAXIS (IP) A PROTECTIVE FACTOR AGAINST GASTRIC CANCER IN IODINE DEFICIENT AREAS?
(1)Szybinski Z, (2)Rachtan J , (1)Huszno B , (1)Hubalewska A , (1)Buziak-Bereza M , (1)Trofimiuk M , (1)Collegium Medicum, Jagiellonian University Chair and Department of Endocrinology Krakow Poland,(2)Oncologic Center M.Sklodowska-Curie s Institute of Oncology Krakow Poland
Epidemiological data reveal an association between gastric cancer incidence rate (GCIR) and iodine deficiency (ID) in endemic goiter areas. The “Venturi hypothesis” is based on the correlation between ID and high GC mortality rate due to common derivation of thyroid gland and stomach from primitive gut. Gastric mucosa and thyroid folliculi can accumulate iodine, have similar enzymes involved in iodine metabolism, are able to iodise tyrosine and express
similar cross-reacting antigens.
The aims of the study were:
1. to compare iodine nutrition after introduction of obligatory IP in 1997 and GCIR
2. to evaluate GCIR in years 1985-1999.

METHODS:
GCIR in Kraków region was calculated using standardized population based register with 100% of histological verification and 85-90% of ascertainment. GCIR was expressed as a number of newly diagnosed cases per 100.000 inhabitants per year. Effectiveness of IP was evaluated by comparative analysis of goiter prevalence and ioduria in schoolchildren carried out in epidemiological survey performed in 1992/1993 and in 1994-2003.

RESULTS Iodine supply on the population level was expressed as a number of tons of iodized household salt on the market: 4000 tones in 1994 and 120.000 tones in 2003. Goiter prevalence in 6-8 years old schoolchildren decreased from 34% in 1992/93 g/L increased from 12% in(to 2,7% in 2003 and frequency of ioduria over 100 1992/1993 to 33-55% in 1999-2003. According to WHO and ICCIDD, Poland is now the country with sufficient iodine supply on the population level. GCIR decreased from 24,2 (92 cases) in 1985-1987 to 17,9 in 1997-1999, all cancers crude incidence rate increased markedly from 273,3 to 380,1 respectively.

CONCLUSIONS:
1. Significant positive correlation between iodine nutrition on the population level and decrease of
GCIR seems to support “Venturi hypothesis” in Poland.
2. Presented data indicate the protective role of IP against gastric cancer.

Source: www.hotthyroidology.com/eta2004/file_info/download1.php?

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3 Responses to “Iodine and Gastric Cancer, Stomach Cancer, 30TH ANNUAL MEETING OF EUROPEAN THYROID ASSOCIATION”

  1. Arrow Durfee Says:

    Technical Iodine info: en.wikipedia.org/wiki/Iodine

  2. Judy Thayer Says:

    Because my husband is on Armour for his thyroid and also has gastritis, I am interested in putting him on iodine and skipping the Armour.

    How much should I give him? Believe me, there is no doctor in the state of Alabama who could give me any direction here.

    Thank you,
    Judy Thayer

  3. Arrow Durfee Says:

    I cannot recommend the dosage but If I were going to attempt this I would read all the literature out on the topic first and much of it is found here on this blog. Go to the top of the page and put iodine in the google search button and make sure you have HealthSalon.org button clicked just below. Articles should come right up for you. Also each article in HealthSalon has a list of related posts at the bottom of the article just before the comments section.

    He for sure will requires to have his thyroid checked about every 6 weeks to make sure his medication levels are ok. Often a reduction is required. Some of the articles will talk about diet also. Make sure he has a good supply of hydrochloric acid. If he is on pepc
    id or other antacids you will have to look into that. HCL is essential for breaking down compounds necessary to form thryoid hormones.

    The need for antacids often occurs due to LACK of hydrochloric acid, not too much. You will have to look into it to understand.